Testimonial

Baby of Anuradha was a case of gastroschisis, was admitted after being refused to be operated on at multiple hospitals in Bilaspur but at Star Children Hospital, The surgical and NICU team did a herculean task of saving this baby and with good cooperation of the parents and precise surgical and tiresome NICU care led to the survival of this very difficult and unique problem which most of the babies don’t survive.
Baby of Anuradha
Parents
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From Misdiagnosis to Full Recovery: A Child’s Battle with MOG Encephalitis

BEFORE
AFTER
  • 1.8 वर्षीय बालिका, जिसे तीव्र आंत्रशोथ था, को जांजगीर में भर्ती कराया गया, उसे गंभीर अम्लीय श्वास (पीएच 6.8) में स्थानांतरित किया गया, आगमन पर कोई न्यूरोलॉजिकल प्रतिक्रिया नहीं हुई (ई1एम1वी1)। हमने मेटाबोलिक एन्सेफैलोपैथी का निदान किया। बच्चे को यांत्रिक वेंटिलेशन की आवश्यकता थी, उसे न्यूरोलॉजिकल रूप से बरकरार रखा गया। टीएमएस रिपोर्ट में उच्च ब्रांच्ड चेन एमिनो एसिड ल्यूसीन/आइसोल्यूसीन और वेलिन का संकेत मिलता है; यह एमएसयूडी या मैलोनिक एसिडेमिया में देखा जा सकता है। दोबारा टीएमएस और जीसीएमएस की योजना बनाई गई है और अधिक सटीक निदान के बाद सटीक दोष का पता लगाने के लिए एक संपूर्ण एक्सोम अनुक्रमण भेजा जाएगा।
  • यह एक दिलचस्प मामला है जिसे मेटाबोलिक एन्सेफैलोपैथी के रूप में प्रस्तुत किया गया था और हम मामले को समय पर पहचान कर उसका निदान और प्रबंधन कर सकते हैं। ऐसे रोगियों को तृतीयक और चतुर्थक पीआईसीयू में समय पर रेफर करना इन रोगियों के जीवन को बचाने में बहुत महत्वपूर्ण है
  • 1.8 year old female child, who had acute gastroenteritis,was admitted in Janjgir, transferred in severe acidotic breathing(pH 6.8), no neurological response at arrival(E1M1V1). We made a diagnosis of metabolic encephalopathy. Child needed mechanical ventilation, discharged neurologically intact. TMS report suggestive of elevated branched chain amino acid leucine/isoleucine and valine; this can be seen in MSUD or Malonic Acidemia. Repeat TMS and GCMS has been planned and following a more precise diagnosis a whole exom sequencing will be sent to detect the exact defect. 
  • This is an interesting case which presented as a metabolic encephalopath
  • ⁠ ⁠10 years old female child admitted with Fever and headache for 7 days, Vomiting for 2 days 5 days back, Pain abdomen for 5 days, Weakness in both legs for 5 days, unable to walk. She was hospitalized for this illness at another hospital for 2 days, provisional diagnosis UTI, was receiving IV antibiotics. As her weakness progressed she had a lumbar puncture done at that hospital as part of her work up which had 25 cells, 70% polymorphs, low sugar. Her referral to us was as a case of meningitis.
    •⁠ ⁠At arrival she had flaccid weakness of both her legs. Power around knee joint, ankle joint was nearly 1/5 MRC grading. MRI Brain was done already at the referring unit. Looking at the MRI and after a detailed neurological evaluation, we suspected MOG encephalitis with spinal cord involvement and an MRI spine was done. MRI Spine confirmed holocord involvement. A repeat CSF was done and samples sent for MOG antibodies to NIMHANS Bangalore. She was started on pulse methyl prednisolone with which she showed improvement. Her pain decreased and limb power started improving. She later on received IVIG and had complete recovery. Her MOG
    antibodies were positive.
    •⁠ ⁠This was a case of Autoimmune Encephalitis diagnosed after suspecting this condition in time. Timely diagnosis and treatment helped the child to regain her walking and prevented further progression of the disease, which could have been lethal.
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